OBJECTIVES: Tricuspid valve (TV) surgery in the adult with congenital heart disease (ACHD) is a frequently performed procedure. The aim of this study was to analyse postoperative and medium-term outcomes. METHODS: We conducted a single-centre retrospective study of patients with ACHD who underwent TV surgery (January 2000-December 2016); patients with Ebstein's anomalies were excluded. Operative and clinical records were reviewed. Outcomes considered were survival, grade of insufficiency/stenosis and TV reoperation at follow-up. RESULTS: A total of 128 patients with ACHD had TV surgery for functional regurgitation (n=95), dysplasia (n=23) and systemic TV (n=10). Median age was 40.8 years [interquartile range (IQR) 25.3]; 55.5% were men. Preoperative regurgitation was classified as mild (n=8), moderate (n=47) and severe (n=70). The TV was repaired in 109 as follows: ring annuloplasty (n=43), de Vega annuloplasty (n=29), Wooler annuloplasty (n=13), commissural plasty (n=9), Kay annuloplasty (n=7) and others (n=8). The TV was replaced in 19 patients with biological (n=10) and mechanical (n=9) prostheses. The median hospital stay was 12 days (IQR 10). The overall mortality rate was 8.6% (n=11): 2 hospital deaths (1.6%) and 9 late deaths. Survival was 93% [95% confidence interval (CI) 85-97%] at 5 years and 83% (95% CI 70-91%) at 10 years. The median follow-up period was 4.95 years (IQR 7.7) with 1 TV reoperation. Echocardiographic assessment showed >= moderate regurgitation in 34 (34.3%) patients. Suture plasty had a significantly higher incidence of TV regurgitation >= moderate compared to ring annuloplasty (48.9% vs 26.3%; P=0.033). CONCLUSIONS: TV surgery in the ACHD is frequently associated with other main procedures. Stabilizing the TV annulus with a prosthetic ring guarantees lower recurrence of moderate to severe regurgitation compared to suture plasty repair.

Results for tricuspid valve surgery in adults with congenital heart disease other than Ebstein's anomaly†

Chessa M;
2019-01-01

Abstract

OBJECTIVES: Tricuspid valve (TV) surgery in the adult with congenital heart disease (ACHD) is a frequently performed procedure. The aim of this study was to analyse postoperative and medium-term outcomes. METHODS: We conducted a single-centre retrospective study of patients with ACHD who underwent TV surgery (January 2000-December 2016); patients with Ebstein's anomalies were excluded. Operative and clinical records were reviewed. Outcomes considered were survival, grade of insufficiency/stenosis and TV reoperation at follow-up. RESULTS: A total of 128 patients with ACHD had TV surgery for functional regurgitation (n=95), dysplasia (n=23) and systemic TV (n=10). Median age was 40.8 years [interquartile range (IQR) 25.3]; 55.5% were men. Preoperative regurgitation was classified as mild (n=8), moderate (n=47) and severe (n=70). The TV was repaired in 109 as follows: ring annuloplasty (n=43), de Vega annuloplasty (n=29), Wooler annuloplasty (n=13), commissural plasty (n=9), Kay annuloplasty (n=7) and others (n=8). The TV was replaced in 19 patients with biological (n=10) and mechanical (n=9) prostheses. The median hospital stay was 12 days (IQR 10). The overall mortality rate was 8.6% (n=11): 2 hospital deaths (1.6%) and 9 late deaths. Survival was 93% [95% confidence interval (CI) 85-97%] at 5 years and 83% (95% CI 70-91%) at 10 years. The median follow-up period was 4.95 years (IQR 7.7) with 1 TV reoperation. Echocardiographic assessment showed >= moderate regurgitation in 34 (34.3%) patients. Suture plasty had a significantly higher incidence of TV regurgitation >= moderate compared to ring annuloplasty (48.9% vs 26.3%; P=0.033). CONCLUSIONS: TV surgery in the ACHD is frequently associated with other main procedures. Stabilizing the TV annulus with a prosthetic ring guarantees lower recurrence of moderate to severe regurgitation compared to suture plasty repair.
2019
Tricuspid valve
Congenital heart disease
Surgery
Outcomes
Adult with congenital heart disease
Valve repair
Long-term outcome
Atrial septal defect
Grown up congenital heart disease
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Utilizza questo identificativo per citare o creare un link a questo documento: https://hdl.handle.net/20.500.11768/117217
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